Mitochondrial reactive oxygen is critical for IL-12/IL-18-induced IFN
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Frontiers Mitochondria, Oxidative Stress and Innate Immunity
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Mitochondrial oxidative stress drives IL-12/IL-18-induced IFN-γ production by CD4+ T cells and is controlled by Fas
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Mitochondrial reactive oxygen is critical for IL-12/IL-18-induced IFN-γ production by CD4+ T cells and is regulated by Fas/FasL signaling
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Mitochondrial reactive oxygen is critical for IL-12/IL-18-induced IFN-γ production by CD4+ T cells and is regulated by Fas/FasL signaling
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Mitochondrial oxidative stress drives IL-12/IL-18-induced IFN-γ production by CD4+ T cells and is controlled by Fas
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